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KMID : 0381219750070090590
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Journal of RIMSK
1975 Volume.7 No. 9 p.590 ~ p.598
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Bronchial Asthma in Childhood
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Abstract
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Bronchial asthma is characterized by recurrent episodic attacks brought about by spasm of bronchiolar smooth muscle mucosal edema, with excessive secretion of thick, tenacious mucus contributes to bronchial obstruction. Bronchial asthma is considered to a hypersensitivity of
bronchial trees which can react to multiple stimuli: specific allergens, e.g., plant pollens, house dust, mold, animal dander (called Extrinsic asthma), or Respiratory infections due to certain varuses (Intrinsic asthma). Infectious asthma occurs more often in infants. Many non-specific s´.imu´.i, e.g., cold, chemicals, exercise, emotions, can act triggering or aggravating factors.
The onset of asthma may be abrupt or insidious. Sudden onsets are often start with a cough or sensation of hunger for air. Expiration is prolonged, wheezing occurs throughout the entire chest. The attack of asthma will not subside spontaneously or promptly if infection is the cause or if the child is continuously exposed to the offenders. The principles of symptomatic treatment of Bronchial asthma are bronchodilation, hydration, relaxation, and environmental control. At attempt to explain the pathogenesis and mechanism of asthma which currently enjoys widespread popularity and has stimulated a great deal of recent investigation is the Beta (ß) Adrenergic theory of Bronchial asthma.
The demonstration that the pertussis induced hypersensitivity state was produced by impaired function of the 9-adrenergic effector system led to the proposal that bronchial asthma in humans might be due to a similar beta adrenergic defect.
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